Inflammation following acute myocardial Infarct . Cellular and molecular interactions involved in cardiac inflammation may give rise to combination therapies.
Microglial involvement in neuropathic pain. Five mechanisms as focal points for assessing opportunities for the development of neuropathic pain therapies.
The link between pain and inflammation. Interactions between the nervous & immune systems and the potential targets that lie in the overlap.
Pain processing and pathways. Choosing suitable behavior tests for common drug classes based on primary mechanism and site of action.